Facial nerve entrapment

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Facial nerve disorders can cause weakness on one or both sides of your face. You might lose your facial expressions, and find it difficult to eat, drink and speak clearly. It can also become difficult to close your eye and blink, which can lead to damage to your cornea.

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One putative mechanism for its occurrence suggests that edematous entrapment of the facial nerve in the meatal foramen the narrowest segment of the internal auditory canal may lead to nerve ischemia or necrosis and subsequent facial nerve dysfunction. To assess whether meatal decompression may help reduce the incidence of delayed facial nerve palsy during microsurgical resection of acoustic tumors, we compared 25 patients undergoing translabyrinthine removal of acoustic neuromas who received prophylactic decompression of the labyrinthine segment of the facial nerve Group 1 with 40 patients who did not receive facial nerve decompression Group 2. No patients in Group 1 had a delayed progressive facial paralysis with degeneration.

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Ulnar nerve entrapment is a condition where the ulnar nerve becomes physically trapped or pinchedresulting in pain, numbness, or weakness. Ulnar neuropathy causes symptoms in a specific anatomic distribution, corresponding parts of the arm innervated by the ulnar nerve:. Motor symptoms consistent of muscle weakness; sensory symptoms or paresthesias consist of numbness or tingling in the areas innervated by the ulnar nerve.

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Facial nerve paralysis is a common problem that involves the paralysis of any structures innervated by the facial nerve. The pathway of the facial nerve is long and relatively convoluted, so there are a number of causes that may result in facial nerve paralysis. Facial nerve paralysis is characterised by facial weakness, usually only in one side of the face, with other symptoms possibly including loss of tastehyperacusis and decreased salivation and tear secretion.

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Facial nerve decompression is a type of nerve decompression surgery where abnormal compression on the facial nerve is relieved. Pressure and compression of any cause on a peripheral nerve can cause nerve impulse block. That is, the nerve is no longer able to send electrochemical impulses, and hence does not send signals to the brain or from the brain to muscles.

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The exact cause is unclear, but believed to involve loss of the myelin around the trigeminal nerve. Treatment includes medication or surgery. It is estimated that 1 in 8, people per year develop trigeminal neuralgia.

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Trigeminal neuralgia is extreme pain and muscle spasms in the face. Attacks of intense, electric shock-like facial pain can occur without warning or be triggered by touching specific areas of the face. Although the exact cause of trigeminal neuralgia is not fully understood, a blood vessel is often found compressing the nerve.

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The facial nerve is one of the key cranial nerves with a complex and broad range of functions. Although at first glance it is the motor nerve of facial expression which begins as a trunk and emerges from the parotid gland as five branches see facial nerve branches mnemonicit has taste and parasympathetic fibers that relay in a complex manner. The facial nerve is the only cranial nerve that may show normal post-contrast enhancement, although this applies only to the labyrinthine segment up to the stylomastoid foramen.

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The cranial nerves are 12 pairs of nerves that emerge from the brain and are responsible for providing motor and sensory functions. They are among the most delicate nerves in the human nervous system and require experts who specialize in their normal and abnormal presentations. Cranial nerve issues can affect a motor nerve, called cranial nerve palsy, or affect a sensory nerve, causing pain or diminished sensation.

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The Facial Nerve pp Cite as. The rationale for facial nerve decompression is to release the facial nerve and its vessels from entrapped compression. We have been investigating the possibility and timing of facial nerve entrapment in the facial canal in the early stages of its damage. We report results of our experiments on the swelling changes of damaged facial nerves with time in guinea pigs [2].

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